https://nova.newcastle.edu.au/vital/access/ /manager/Index ${session.getAttribute("locale")} 5 https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:35919 Wed 29 Jan 2020 13:18:20 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:50112 Wed 28 Feb 2024 16:20:34 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:54521 Wed 28 Feb 2024 16:15:39 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:53886 Wed 28 Feb 2024 14:50:48 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:50381 Wed 28 Feb 2024 14:46:50 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:39547 Wed 27 Jul 2022 14:28:27 AEST ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:45158 N = 70) who had experienced a stroke between 5 months and 28 years ago were included in the cross-sectional study, along with age-matched controls (N = 70). We measured stress using both the Perceived Stress Scale and biological markers, and resilience using both the Brief Resilience Scale and the Connor-Davidson Resilience Scale. Stroke outcomes were assessed using the Stroke Impact Scale. We found that, compared with age-matched controls, stroke survivors reported greater levels of perceived stress, and lower levels of resilience. In stroke survivors, both perceived stress and resilience were independently associated with stroke outcomes in linear regression models. In particular, these relationships were observed for cognitive outcomes including mood, memory, and communication. The association between stress and stroke outcome did not differ across time post stroke. Given that resilience is a modifiable psychological construct, future research may consider whether strategies directed at enhancing resilience may improve recovery from stroke. Australia and New Zealand Clinical Trials Registry: ACTRN12617000736347.]]> Wed 26 Oct 2022 19:17:21 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:33344 Wed 24 Nov 2021 15:52:58 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:38259 Wed 22 Mar 2023 17:26:27 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:36269 Wed 18 Mar 2020 15:29:31 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:35239 Wed 17 Nov 2021 16:31:11 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:53778 Wed 13 Mar 2024 19:07:43 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:38833 Wed 13 Mar 2024 13:57:46 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:53828 Wed 13 Mar 2024 13:45:57 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:48045 Wed 13 Mar 2024 13:43:43 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:51756 Wed 13 Mar 2024 07:52:48 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:51966 Wed 13 Mar 2024 07:44:19 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:21634 Wed 11 Apr 2018 17:13:00 AEST ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:26096 Wed 11 Apr 2018 16:27:54 AEST ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:23556 Wed 11 Apr 2018 16:11:40 AEST ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:22445 Wed 11 Apr 2018 16:04:55 AEST ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:26974 Wed 11 Apr 2018 13:56:47 AEST ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:29331 Wed 11 Apr 2018 12:30:03 AEST ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:21983 Wed 11 Apr 2018 12:06:20 AEST ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:29423 Wed 11 Apr 2018 11:56:16 AEST ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:26247 Wed 11 Apr 2018 11:24:34 AEST ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:23520 Wed 11 Apr 2018 09:55:01 AEST ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:31256 Wed 11 Apr 2018 09:48:38 AEST ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:39228 Wed 10 Aug 2022 08:45:34 AEST ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:32912 Wed 09 Mar 2022 16:02:02 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:33101 Wed 09 Feb 2022 15:58:21 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:47411 Wed 06 Mar 2024 15:04:55 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:48387 Wed 06 Mar 2024 14:34:39 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:33361 Wed 04 Sep 2019 12:18:18 AEST ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:30359 Wed 04 Sep 2019 09:47:44 AEST ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:38160 0.05), and the intervention significantly improved the ASI (all p < 0.05). A more favourable ASI score was associated with improved symptoms of depression, anxiety, stress, QOL-mental and of energy and fatigue (all p < 0.05). The intervention effects on symptoms of depression ([AB; 95%CI] -0.31; - 0.60,-0.11), anxiety (- 0.11; - 0.27,-0.01), stress (- 0.37; - 0.65,-0.174), QOL-mental (0.53; 0.22, 1.01) and ratings of energy and fatigue (0.85; 0.33, 1.63) were mediated by ASI. At 6 months the magnitude of association was larger although the overall pattern of results remained similar. Conclusions: Improvements in the overall physical activity and sleep behaviours of adults partially mediated the intervention effects on mental health and quality of life outcomes. This highlights the potential benefit of improving the overall pattern of physical activity and sleep on these outcomes. Trial registration: Australian New Zealand Clinical Trial Registry: ACTRN12617000680369; ACTRN12617000376347. Universal Trial number: U1111–1194-2680; U1111–1186-6588. Human Research Ethics Committee Approval: H-2016- 0267; H-2016–0181.]]> Wed 04 Aug 2021 18:44:37 AEST ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:45650 Wed 02 Nov 2022 15:31:05 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:36341 Wed 01 Apr 2020 15:20:40 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:16906 A agonist muscimol or saline into the amygdala bilaterally and were subjected to a respiratory recording using whole body plethysmography. Presentation of acoustic stimuli (500-ms white noise, 40–90 dB) caused transient responses in respiratory rate that were proportional to the stimulus intensity, ranging from +13 ± 9 cpm to +276 ± 67 cpm for 40- and 90-dB stimuli, respectively. Inhibition of the amygdala significantly suppressed respiratory rate responses to the high-intensity stimuli (70–90dB). Submitting rats to the restraint stress significantly elevated the mean respiratory rate (+72 ± 8 cpm) and the dominant respiratory rate (+1 ± 12 cpm), as well as the fraction of high-frequency respiratory rate (+10 ± 3%). Inhibition of the amygdala by muscimol significantly suppressed these responses. We conclude that the amygdala is one of the key structures that are essential for expression of respiratory responses to stressful or alerting stimuli in rats.]]> Tue 31 Jul 2018 15:18:15 AEST ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:55205 Tue 30 Apr 2024 10:50:20 AEST ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:38070 Tue 21 Mar 2023 09:26:38 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:41940 n = 478) or without (n = 8134) a self-reported diagnosis of PCOS. Main outcome measures were depression, anxiety and perceived stress measured using validated scales. The χ² and t tests were used to assess differences between groups. Univariable and multivariable regression were performed to determine factors contributing to each outcome. Results: Women reporting PCOS, compared with women not reporting PCOS, reported higher prevalence of depression (27.3% v. 18.8%), anxiety symptoms (50% v. 39.2%) and greater score for perceived stress (1.01 ± 0.03 v. 0.88 ± 0.01). After adjusting for body mass index, infertility and socio-demographic factors, women with PCOS were still more likely to be depressed, anxious and to have a higher level of perceived stress. There was a high-level mediation effect of stress between PCOS and both depression and anxiety. Conclusion: Compared with women not reporting PCOS, women reporting PCOS have increased depression, anxiety and perceived stress. Stress may play a role in the association between PCOS, depression and anxiety. Further studies should consider assessment and management of stress in PCOS as it may be relevant for understanding the aetiology and treatment of psychological distress.]]> Tue 16 Aug 2022 10:54:17 AEST ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:54240 Tue 13 Feb 2024 13:19:59 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:45896 Tue 08 Nov 2022 09:04:05 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:23554 Tue 08 May 2018 16:17:57 AEST ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:30749 Tue 03 Sep 2019 18:26:17 AEST ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:34748 Tue 03 Sep 2019 18:19:01 AEST ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:30659 Tue 01 May 2018 08:52:05 AEST ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:39914 Thu 30 Jun 2022 11:34:33 AEST ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:33592 Auricularia polytricha is a popular mushroom found all over the world. In this study we considered the effect of an aqueous extract of A. polytricha (AEAP) on restoring sexual performance parameters to normal, evaluated by considering observations of sexual behavior. At 0, 6, 12, 18, and 24 days, the following parameters of sexual performance were identified before and throughout the observations: mount latency, intromission latency, ejaculation latency, mounting frequency, intromission frequency, ejaculation frequency, and postejaculatory interval. Treatment of rats under stress with AEAP showed promising effects on overcoming stress-induced sexual dysfunction, on sexual performance, and on accessory sexual organs and body weight. Mounting latency, intromission latency, ejaculation latency, and postejaculatory interval parameters were significantly decreased by AEAP, whereas mounting frequency, intromission frequency, and ejaculation frequency were significantly increased by AEAP. These properties were identified in sexually dynamic and indolent male rats. We conclude that AEAP has a potent aphrodisiac activity.]]> Thu 22 Nov 2018 13:41:22 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:35914 Thu 16 Jan 2020 14:56:08 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:43297 Thu 15 Sep 2022 12:51:33 AEST ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:54278 Thu 15 Feb 2024 14:38:24 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:27474 Thu 13 Sep 2018 16:54:06 AEST ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:44419 Thu 13 Oct 2022 09:51:45 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:21637 Thu 11 Jul 2019 14:47:53 AEST ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:36885 Thu 04 Nov 2021 10:39:34 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:50738 Thu 03 Aug 2023 16:40:32 AEST ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:49117 Thu 02 May 2024 12:18:23 AEST ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:47846 Thu 02 Feb 2023 15:57:49 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:35193 Thu 01 Aug 2019 16:25:07 AEST ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:31666 How are you? I am totally stressed at the moment, to be honest. Work is piling up and I’m just drowning. I don’t know when I’m going to have time to start on that secrecy and silence book chapter – I’m so, so late with it now, and I feel really bad that I’m letting Roisin down, but I literally never have a second. I know, I know exactly what you mean. I mean, I had 115 e-mails yesterday and they all needed answering. I’m doing 16 hour days just trying to keep on top of it. I feel like I’m always late with everything, and my ›to do‹ list grows faster than I can cross things off it. It’s like one of those fungi in a horror movie that doubles in size every few hours! (Laughter) And I never ever have chance to do any of my own work. I’m sleeping really badly and it all just feels completely out of control...It’s the same for me. Reading? What that? Thinking? No chance! And you feel awful, don’t you. With me I feel like I’m constantly stealing time from the kids too – I’ll go off to check messages in the middle of a game of Monopoly or something. Sometimes I just feel like quitting. Yeah I know. It just gets worse. Still hoping to win the lottery, then?(laughter) But how are you? Do you really want to know?! (laughter) (Yeh) well, awful actually. I’m really fed up. I heard yesterday that my article for x journal was turned down. (Oh no!) You know, the one I worked on for ages and ages. I poured so much of myself into that piece (I know). And one of the referee’s comments was vile – it said something like ›my first year undergraduates have a better understanding of the field than this author does – why are they wasting all of our time‹. When I read it it was like a slap in the face, Ros. It was all I could do not to burst out crying in the postroom, but I had a lecture right afterwards so I somehow managed to pull myself together and go and do that. But last night, I just didn’t sleep (poor you) I just kept on going over and over with all these negative comments ringing round my head. And you know the worst thing is, they are right: I am useless (no you’re not), I’m a complete fraud, and I should have realised that I was going to be found out if I sent my work to a top journal like that. This is a transcript of a conversation I had with a female friend in the few days before (finally) beginning work on this chapter. Both speakers are white, both work in »old« (pre-1992) British Universities, and both are employed on »continuing« contracts – thus are already marked as »privileged« in multiple ways in the contemporary academy. Mine is easily recognizable as the voice which worries about how late this article is! Some readers may find this fragment of conversation rather odd, but I suspect for many more it will appear familiar and may strike deep chords of recognition. It speaks of many things: exhaustion, stress, overload, insomnia, anxiety, shame, aggression, hurt, guilt and feelings of out-of-placeness, fraudulence and fear of exposure within the contemporary academy. These feelings, these affective embodied experiences, occupy a strange position in relation to questions of secrecy and silence. They are at once ordinary and everyday, yet at the same time remain largely secret and silenced in the public spaces of the academy. They are spoken in a different, less privileged register; they are the stuff of the chat in the corridor, coffee break conversations and intimate exchanges between friends, but not, it would seem, the keynote speech or the journal publication or even the departmental meeting. For all the interest in reflexivity in recent decades, the experiences of academics have somehow largely escaped critical attention. It is as if the parameters for reflexivity are bounded by the individual study, leaving the institutional context in which academic knowledge is produced simply as a taken for granted backdrop.]]> Sat 24 Mar 2018 08:44:53 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:31671 Sat 24 Mar 2018 08:44:53 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:20933 Sat 24 Mar 2018 08:06:05 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:30350 Sat 24 Mar 2018 07:40:25 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:27703 Sat 24 Mar 2018 07:40:10 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:28082 Sat 24 Mar 2018 07:39:48 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:28456 Sat 24 Mar 2018 07:39:30 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:29566 Sat 24 Mar 2018 07:37:54 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:30841 Sat 24 Mar 2018 07:33:55 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:26380 Sat 24 Mar 2018 07:33:04 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:29502 Sat 24 Mar 2018 07:29:45 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:28273 Sat 24 Mar 2018 07:28:31 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:26422 Sat 24 Mar 2018 07:27:59 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:30871 Sat 24 Mar 2018 07:26:39 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:30574 Sat 24 Mar 2018 07:23:57 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:27592 Sat 24 Mar 2018 07:23:41 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:22180 Sat 24 Mar 2018 07:16:25 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:24495 2+ and ROS waves), chemical (assimilates, hormones, peptides and nutrients), and molecular (proteins and RNA) signals. Further, different signalling systems operate at very different timescales. It remains unclear whether some of these signalling systems operate in a priming mode(s), whereas others deliver more specific information about the nature of the signal, or whether they carry the same 'weight'. This review summarises the current knowledge of the above signalling mechanisms, and reveals their hierarchy, and highlights the importance of integration of these signalling components, to enable optimal plant functioning in a dynamic environment.]]> Sat 24 Mar 2018 07:13:15 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:24042 Sat 24 Mar 2018 07:11:12 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:24079 A agonist muscimol on respiration during stress and presentation of brief alerting stimuli in conscious unrestrained rats. We found that integrity of the dlPAG is not essential for stress-induced increase in basal/resting respiratory rate or for generation of respiratory responses to brief alerting stimuli. However, blockade of the dlPAG reduced the amount of motor activity and concomitant high-frequency respiratory activity during restraint and the first 5 min of novelty stress. We conclude that the integrity of the dlPAG is not essential for generation of respiratory component of the defense reaction, but it mediates expression of the fight-or-flight response including its respiratory component.]]> Sat 24 Mar 2018 07:09:37 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:48169 Sat 11 Mar 2023 12:22:50 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:53155 Vglut2+) to the medial insular cortex in this behavior. Whole brain activation mapping and hierarchical clustering of Euclidean distances revealed distinct patterns of coactivation unique to stress-induced binge eating. At a pathway-specific level, PVT^Vglut2+ cells projecting to the medial insular cortex were specifically activated in response to stress-induced binge eating. Subsequent chemogenetic inhibition of this pathway suppressed stress-induced binge eating. We have identified a distinct PVT^Vglut2+ to insular cortex projection as a key driver of "emotional" stress-induced binge eating in female mice, highlighting a novel circuit underpinning this sex-specific behavior.]]> Mon 27 Nov 2023 15:54:48 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:32750 Mon 23 Sep 2019 13:16:28 AEST ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:32683 Mon 23 Sep 2019 12:37:34 AEST ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:37779 4) transporters. PHO2 is itself under tight regulation by the PO₄ responsive microRNA (miRNA), miR399. The DOUBLE-STRANDED RNA BINDING (DRB) proteins, DRB1, DRB2 and DRB4, have each been assigned a specific functional role in the Arabidopsis thaliana (Arabidopsis) miRNA pathway. Here, we assessed the requirement of DRB1, DRB2 and DRB4 to regulate the miR399/PHO2 expression module under PO₄ starvations conditions. Via the phenotypic and molecular assessment of the knockout mutant plant lines, drb1, drb2 and drb4, we show here that; (1) DRB1 and DRB2 are required to maintain P homeostasis in Arabidopsis shoot and root tissues; (2) DRB1 is the primary DRB required for miR399 production; (3) DRB2 and DRB4 play secondary roles in regulating miR399 production, and; (4) miR399 appears to direct expression regulation of the PHO2 transcript via both an mRNA cleavage and translational repression mode of RNA silencing. Together, the hierarchical contribution of DRB1, DRB2 and DRB4 demonstrated here to be required for the appropriate regulation of the miR399/PHO2 expression module identifies the extreme importance of P homeostasis maintenance in Arabidopsis to ensure that numerous vital cellular processes are maintained across Arabidopsis tissues under a changing cellular environment.]]> Mon 19 Apr 2021 11:56:05 AEST ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:40790 Mon 18 Jul 2022 16:21:19 AEST ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:41915 14). Limitations: The lack of a control group and the absence of a complete clinical assessment at the follow-up limit the interpretability of our results. Conclusions: Our data driven analysis suggest insomnia heterogeneity can be categorized into sub-classes by depression, anxiety, and stress symptoms. In addition, insomnia patients with stress and depression symptoms maintained highest percentage of clinical depression at the end-of-treatment and insomnia at follow-up, in comparison with others classes. Stress and depression symptoms should be considered risk factors that play an important role in the long-term outcome of CBT-I.]]> Mon 15 Aug 2022 16:00:33 AEST ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:46279 Mon 14 Nov 2022 15:35:09 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:46203 Mon 14 Nov 2022 11:29:55 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:38647 Mon 06 Dec 2021 16:41:16 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:34336 Mon 04 Mar 2019 12:02:55 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:32599 Mon 03 Jun 2019 16:57:22 AEST ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:50557 Fri 28 Jul 2023 10:53:26 AEST ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:40655 Fri 22 Jul 2022 14:02:02 AEST ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:36620 Fri 22 Apr 2022 10:19:51 AEST ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:37144 Fri 21 Aug 2020 12:35:43 AEST ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:53151 Fri 17 Nov 2023 12:17:06 AEDT ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:51698 Fri 15 Sep 2023 10:31:50 AEST ]]> https://nova.newcastle.edu.au/vital/access/ /manager/Repository/uon:46119 N = 8,177) were followed up every 2 to 3 years to ages 37 to 42 using the Australian Longitudinal Study on Women's Health. A latent class analysis identified obesity-risk classes based on time-dependent measures of income, education, physical activity, sleep quality, dietary behavior, depression, stressful events, and social functioning. Cox proportional hazards regression models examined associations between incident obesity and latent classes, controlling for covariates. Results: Four latent classes were identified, including a lower-risk referent class and classes characterized by poor exercise and diet, stress and low income, and multiple intermediate-level risks. Compared with the referent, all three risk classes had significantly higher obesity risk, with the highest risk in the stress and low-income group (hazard ratio = 2.22; 95% CI: 1.92-2.56). Individual risks associated with obesity included lower education, stressful life events, and lower vigorous physical activity. Conclusions: Young women without baseline obesity were at risk of developing obesity when they experienced co-occurring behavioral, socioeconomic, and psychosocial risks. Both latent classes and individual risk indicators offer insights into prevention strategies.]]> Fri 11 Nov 2022 15:01:11 AEDT ]]>